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الثلاثي التشخيصي الكلاسيكي لالتهاب البنكرياس المزمن


الثلاثي التشخيصي الكلاسيكي لالتهاب البنكرياس المزمن

المعلومة

الثلاثي التشخيصي الكلاسيكي لالتهاب البنكرياس المزمن هو :
تكلسات بنكرياسية Pancreatic calcification
داء سكري Diabetes
إسهال دهني Steatorrhea

المرجع

MedStudy

Dr.Hiba's picture
by
طالب دراسات عليا


شكرا هبة .
اذا ولد ضغير ومعو التهاب بنكرياس , شو بدو يكون السبب ؟

qusei

Quote:
اذا ولد ضغير ومعو التهاب بنكرياس , شو بدو يكون السبب ؟

التليف الكيسي Cystic fibrosis على الأغلب....

Fouad's picture
Fouad
بعد التخرج


أو تشوه خلقي وبعتقد هو الأهم

moonberg's picture
moonberg
بعد التخرج

Quote:
أو تشوه خلقي وبعتقد هو الأهم

قصدك متل البنكرياس المنقسم Pancreas divisum؟؟

Fouad's picture
Fouad
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هو دا....عم حاول أتذكرو ونسيت شكرا فؤاد:razz: Razz

moonberg's picture
moonberg
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Quote:
التليف الكيسي Cystic fibrosis على الأغلب....

تماما هو الاشيع من Pancreas divisum

qusei

Hallo men

I'm sorry but I hope to be sure of ur information coz the cystic fibrosis causes pancreas insufficiency not pancreatitis
and this is from e-medicine
Pancreas

As a part of normal digestion, stomach acid is neutralized by pancreatic bicarbonate, leading to the optimal pH for pancreatic enzyme action. Reduced bicarbonate secretion in response to secretin stimulation has been demonstrated in patients with CF with both PI and pancreatic sufficiency (PS). Reduced bicarbonate secretion affects the digestion so that neither endogenous nor exogenous pancreatic enzymes can work at their optimal pH.

Other factors, such as reduction of water content of secretions, precipitation of proteins, and plugging of ductules and acini, prevent the pancreatic enzymes from reaching the gut. Autodigestion of the pancreas occasionally leads to pancreatitis. Most patients with CF (90-95%) have pancreatic enzyme insufficiency and present with digestive symptoms and/or failure to thrive early in life. However, onset of PI varies and may occur in patients older than 6 months.

Some patients never develop PI. Patients with PI typically present with poor weight gain in association with frequent stools that are malodorous, greasy, and associated with flatulence and colicky pain after feeding. The combination of increased energy intake demand at baseline, the added energy intake demand of chronic disease, difficulty sustaining energy uptake because of malabsorption, and anorexia associated with ongoing lung inflammation leads to poor weight gain. PI predisposes patients to poor absorption of fat-soluble vitamins A, D, E, and K. Symptomatic deficiency of any of these vitamins can occur before diagnosis or as a later complication of the disease.

moonberg's picture
moonberg
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